![]() Gao, Zhenyan Ying, Xiaolan Yan, Jin Wang, Ju Cai, Shizhong Yan, Chonghuai PMID:24888360Īcute mercury vapor poisoning in a 3-month-old infant: A case report. Natural biologic scavengers such as algae, azolla and other aquatic plants possess the ability to uptake mercury traces from the environment. succimer) and dimercaptopropanesulfoxid acid (DMPS) are currently used as chelating agents in mercury poisoning. Besides supportive therapy, British anti lewisite, dimercaprol (BAL), 2,3-dimercaptosuccinic acid (DMSA. Silver nanoparticles are used as a sensitive detector of low concentration Hg2+ ions in homogeneous aqueous solutions. Gold nanoparticles (AuNPs) are a rapid, cheap and sensitive method for detection of thymine bound mercuric ions. methyl mercury) can be detected easily in feces. Urinary concentration is a good indicator of poisoning of elemental and inorganic mercury, but organic mercury (e.g. Mercury level can be measured in plasma, urine, feces and hair samples. water, food, soil and air lead to toxic effects on cardiovascular, pulmonary, urinary, gastrointestinal, neurological systems and skin. Long term exposure to mercury compounds from different sources e.g. ![]() Mercury compounds are classified in different chemical types such as elemental, inorganic and organic forms. Mercury poisoning cases have been reported in many parts of the world, resulting in many deaths every year. As mercury-containing cosmetic products can contaminate the home, some close household contacts were also reported to haveĬurrent approaches of the management of mercury poisoning: need of the hour Nephrotic syndrome (mainly due to minimal change or membranous nephropathy) and neurotoxicity were the most common presenting features. Inorganic mercury poisoning following the use of skin-lightening creams has been reported from Africa, Europe, USA, Mexico, Australia and Hong Kong. Although penetration of the blood-brain barrier by inorganic mercury is poor, prolonged exposure can result in central nervous system (CNS) accumulation and neurotoxicity. Gastrointestinal symptoms include a metallic taste, gingivostomatitis, nausea and hypersalivation. ![]() The kidneys are the major site of inorganic mercury deposition renal damage includes reversible proteinuria, acute tubular necrosis and nephrotic syndrome. The half-life is approximately 1-2 months. With long-term exposure, urinary excretion is the major route of elimination. After absorption, inorganic mercury is distributed widely and elimination occurs primarily through the urine and feces. Ingestion may occur after topical application around the mouth and hand-to-mouth contact. The degree of dermal absorption varies with the skin integrity and lipid solubility of the vehicle in the cosmetic products. The rate of dermal absorption increases with the concentration of mercury and prior hydration of the skin. These searches identified 118 citations of which 31 were relevant. Reports of skin-lightening cosmetic products containing mercury and cases of mercury poisoning following the use of such products were identified using Medline (1950 - 28 March 2011) with mercury, mercury compounds, mercury poisoning, cosmetics and skin absorption as the subject headings. Yet, the public continue to have access to these products. Mercury and mercury salts, including mercurous chloride and mercurous oxide, are prohibited for use in cosmetic products as skin-lightening agents because of their high toxicity. Inorganic mercury poisoning associated with skin-lightening cosmetic products. The Taki Onqoy in 16th-century Peru represents the largest epidemic of mercury poisoning known to humanity. The Cachexia mercurial, the last stage of the disease, would be associated with the idea of pishtaco. It appeared as a result of the exploitation of the mercury mines of Paras and Huancavelica in Peru during the 16th century. Taki Onqoy is a syndrome that corresponds to the poisoning from exposure to mercury. It is suggested that if penicillamine D is used in the treatment of mercury poisoning, steroid cover should be given from the start. Little improvement followed a switch to B.A.L. They were first treated with penicillamine D, but in all but one case this had to be stopped because of rashes. The clinical findings in the affected workers are described. Of 25 workers exposed, seven developed serious chronic mercury poisoning and were admitted to hospital. In a factory in which old aeroplane batteries containing zinc mercury amalgam were substituted, it was not realized that mercury vapour was being produced. In the manufacture of zinc oxide the raw material normally used in zinc metal plates. Mercury Poisoning from an Unsuspected Source
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